Cx43-Dependent Skeletal Phenotypes Are Mediated by Interactions between the Hapln1a-ECM and Sema3d during Fin Regeneration.
Skeletal development is a tightly regulated process and requires proper communication between the cells for efficient exchange of information. Analysis of fin length mutants has revealed that the gap junction protein Connexin43 (Cx43) coordinates cell proliferation (growth) and joint formation (patt...
Main Authors: | Jayalakshmi Govindan, Kyaw Min Tun, M Kathryn Iovine |
---|---|
Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2016-01-01
|
Series: | PLoS ONE |
Online Access: | http://europepmc.org/articles/PMC4734779?pdf=render |
Similar Items
-
Hapln1a is required for connexin43-dependent growth and patterning in the regenerating fin skeleton.
by: Jayalakshmi Govindan, et al.
Published: (2014-01-01) -
The Cx43-like connexin protein Cx40.8 is differentially localized during fin ontogeny and fin regeneration.
by: Sarah V Gerhart, et al.
Published: (2012-01-01) -
Cardiac Cx43 and ECM Responses to Altered Thyroid Status Are Blunted in Spontaneously Hypertensive versus Normotensive Rats
by: Matus Sykora, et al.
Published: (2019-08-01) -
Smart ECM-Based Electrospun Biomaterials for Skeletal Muscle Regeneration
by: Sara Politi, et al.
Published: (2020-09-01) -
Functional study of mutant Cx29, Cx43 and pseudo-Cx43 in prelingual non-syndromic sensorineural deafness
by: Huei Mei, et al.
Published: (2010)