Pain-related increase of excitatory transmission and decrease of inhibitory transmission in the central nucleus of the amygdala are mediated by mGluR1
<p>Abstract</p> <p>Neuroplasticity in the central nucleus of the amygdala (CeA), particularly its latero-capsular division (CeLC), is an important contributor to the emotional-affective aspects of pain. Previous studies showed synaptic plasticity of excitatory transmission to the C...
Main Authors: | Neugebauer Volker, Ren Wenjie |
---|---|
Format: | Article |
Language: | English |
Published: |
SAGE Publishing
2010-12-01
|
Series: | Molecular Pain |
Online Access: | http://www.molecularpain.com/content/6/1/93 |
Similar Items
-
Enhanced group II mGluR-mediated inhibition of pain-related synaptic plasticity in the amygdala
by: Bird Gary C, et al.
Published: (2006-05-01) -
Homer1a signaling in the amygdala counteracts pain-related synaptic plasticity, mGluR1 function and pain behaviors
by: Kuner Rohini, et al.
Published: (2011-05-01) -
mGluR-LTD at Excitatory and Inhibitory Synapses in the Lateral Habenula Tunes Neuronal Output
by: Kristina Valentinova, et al.
Published: (2016-08-01) -
Effects of nitric oxide on excitatory and inhibitory transmission in the nucleus tractus solitarius
by: Wang, Sheng
Published: (2006) -
mGluR1 receptors contribute to non-purinergic slow excitatory transmission to submucosal VIP neurons of guinea-pig ileum
by: Jaime Pei Pei Foong, et al.
Published: (2009-05-01)