Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.

[This corrects the article DOI: 10.1371/journal.ppat.1006703.].

Bibliographic Details
Main Authors:	Romel D Mackelprang, Michael J Bamshad, Jessica X Chong, Xuanlin Hou, Kati J Buckingham, Kathryn Shively, Guy deBruyn, Nelly R Mugo, James I Mullins, M Juliana McElrath, Jared M Baeten, Connie Celum, Mary J Emond, Jairam R Lingappa, Partners in Prevention HSV/HIV Transmission Study and the Partners PrEP Study Teams
Format:	Article
Language:	English
Published:	Public Library of Science (PLoS) 2019-02-01
Series:	PLoS Pathogens
Online Access:	https://doi.org/10.1371/journal.ppat.1007588

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spelling	doaj-379d5f91301d46ba831379390e3ac1662021-04-21T17:11:51ZengPublic Library of Science (PLoS)PLoS Pathogens1553-73661553-73742019-02-01152e100758810.1371/journal.ppat.1007588Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.Romel D MackelprangMichael J BamshadJessica X ChongXuanlin HouKati J BuckinghamKathryn ShivelyGuy deBruynNelly R MugoJames I MullinsM Juliana McElrathJared M BaetenConnie CelumMary J EmondJairam R LingappaPartners in Prevention HSV/HIV Transmission Study and the Partners PrEP Study Teams[This corrects the article DOI: 10.1371/journal.ppat.1006703.].https://doi.org/10.1371/journal.ppat.1007588
collection	DOAJ
language	English
format	Article
sources	DOAJ
author	Romel D Mackelprang Michael J Bamshad Jessica X Chong Xuanlin Hou Kati J Buckingham Kathryn Shively Guy deBruyn Nelly R Mugo James I Mullins M Juliana McElrath Jared M Baeten Connie Celum Mary J Emond Jairam R Lingappa Partners in Prevention HSV/HIV Transmission Study and the Partners PrEP Study Teams
spellingShingle	Romel D Mackelprang Michael J Bamshad Jessica X Chong Xuanlin Hou Kati J Buckingham Kathryn Shively Guy deBruyn Nelly R Mugo James I Mullins M Juliana McElrath Jared M Baeten Connie Celum Mary J Emond Jairam R Lingappa Partners in Prevention HSV/HIV Transmission Study and the Partners PrEP Study Teams Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1. PLoS Pathogens
author_facet	Romel D Mackelprang Michael J Bamshad Jessica X Chong Xuanlin Hou Kati J Buckingham Kathryn Shively Guy deBruyn Nelly R Mugo James I Mullins M Juliana McElrath Jared M Baeten Connie Celum Mary J Emond Jairam R Lingappa Partners in Prevention HSV/HIV Transmission Study and the Partners PrEP Study Teams
author_sort	Romel D Mackelprang
title	Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.
title_short	Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.
title_full	Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.
title_fullStr	Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.
title_full_unstemmed	Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.
title_sort	correction: whole genome sequencing of extreme phenotypes identifies variants in cd101 and ube2v1 associated with increased risk of sexually acquired hiv-1.
publisher	Public Library of Science (PLoS)
series	PLoS Pathogens
issn	1553-7366 1553-7374
publishDate	2019-02-01
description	[This corrects the article DOI: 10.1371/journal.ppat.1006703.].
url	https://doi.org/10.1371/journal.ppat.1007588
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Correction: Whole genome sequencing of extreme phenotypes identifies variants in CD101 and UBE2V1 associated with increased risk of sexually acquired HIV-1.

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