Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis

Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis

<p>Abstract</p> <p>Background</p> <p>Myelin Oligodendrocyte Glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE) is the most commonly used mouse model for multiple sclerosis (MS). During the of progression of EAE, microglia, the immunocompetent cel...

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Bibliographic Details
Main Authors:	Tsirka Stella E, Wu Muzhou, Bhasin Madhuri
Format:	Article
Language:	English
Published:	BMC 2007-07-01
Series:	BMC Immunology
Online Access:	http://www.biomedcentral.com/1471-2172/8/10

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