Erythromycin Suppresses the Cigarette Smoke Extract-Exposed Dendritic Cell-Mediated Polarization of CD4+ T Cells into Th17 Cells
Cigarette smoke is a major effector of chronic obstructive pulmonary disease (COPD), and Th17 cells and dendritic cells (DCs) involve in the pathogenesis of COPD. Previous studies have demonstrated the anti-inflammatory effects of macrolides. However, the effects of macrolides on the cigarette smoke...
Main Authors: | Jifeng Liu, Xiaoning Zhong, Zhiyi He, Jianquan Zhang, Jing Bai, Guangnan Liu, Yi Liang, Leilei Ya, Xianglin Qin |
---|---|
Format: | Article |
Language: | English |
Published: |
Hindawi Limited
2020-01-01
|
Series: | Journal of Immunology Research |
Online Access: | http://dx.doi.org/10.1155/2020/1387952 |
Similar Items
-
Effect of cigarette smoke extract on dendritic cells and their impact on T-cell proliferation.
by: Esmaeil Mortaz, et al.
Published: (2009-01-01) -
Cigarette Smoke Decreases the Maturation of Lung Myeloid Dendritic Cells.
by: Elena Arellano-Orden, et al.
Published: (2016-01-01) -
Effects of Low-Dose and Long-Term Treatment with Erythromycin on Interleukin-17 and Interleukin-23 in Peripheral Blood and Induced Sputum in Patients with Stable Chronic Obstructive Pulmonary Disease
by: Caimei Tan, et al.
Published: (2016-01-01) -
Effect of roflumilast on inflammatory cells in the lungs of cigarette smoke-exposed mice
by: De Cunto Giovanna, et al.
Published: (2008-08-01) -
Identification of key genes in human airway epithelial cells in response to respiratory pathogens using microarray analysis
by: Yinghua Li, et al.
Published: (2018-06-01)