The ataxia (axJ) mutation causes abnormal GABAA receptor turnover in mice.

The ataxia (axJ) mutation causes abnormal GABAA receptor turnover in mice.

Ataxia represents a pathological coordination failure that often involves functional disturbances in cerebellar circuits. Purkinje cells (PCs) characterize the only output neurons of the cerebellar cortex and critically participate in regulating motor coordination. Although different genetic mutatio...

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Main Authors: Corinna Lappe-Siefke, Sven Loebrich, Wulf Hevers, Oliver B Waidmann, Michaela Schweizer, Susanne Fehr, Jean-Marc Fritschy, Ivan Dikic, Jens Eilers, Scott M Wilson, Matthias Kneussel
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2009-09-01
Series:PLoS Genetics
Online Access:http://europepmc.org/articles/PMC2744266?pdf=render

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