On the mechanism of synaptic depression induced by CaMKIIN, an endogenous inhibitor of CaMKII.

On the mechanism of synaptic depression induced by CaMKIIN, an endogenous inhibitor of CaMKII.

Activity-dependent synaptic plasticity underlies, at least in part, learning and memory processes. NMDA receptor (NMDAR)-dependent long-term potentiation (LTP) is a major synaptic plasticity model. During LTP induction, Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) is activated, autophospho...

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Bibliographic Details
Main Authors: Camilo Gouet, Belen Aburto, Cecilia Vergara, Magdalena Sanhueza
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2012-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC3493544?pdf=render

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