The Ubr2 gene is expressed in skeletal muscle atrophying as a result of hind limb suspension, but not Merg1a expression alone

The Ubr2 gene is expressed in skeletal muscle atrophying as a result of hind limb suspension, but not Merg1a expression alone

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Skeletal muscle (SKM) atrophy is a potentially debilitating condition induced by muscle disuse, denervation, many disease states, and aging. The ubiquitin proteasome pathway (UPP) contributes greatly to the protein loss suffered in muscle atrophy. The MERG1a K+ channel is known to induce UPP activit...

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Bibliographic Details
Main Authors: Gregory H. Hockerman, Nicole M. Dethrow, Sohaib Hameed, Maureen Doran, Christine Jaeger, Wen-Horng Wang, Amber L. Pond
Format: Article
Language:English
Published: PAGEPress Publications 2014-09-01
Series:European Journal of Translational Myology
Subjects:
Skeletal muscle atrophy
UBR2
E3-II
ERG1a potassium channel
hind limb suspension
E3 ligase
ubiquitin proteasome pathway
Online Access:http://www.pagepressjournals.org/index.php/bam/article/view/3319

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