Receptor for Advanced Glycation End Products and its Inflammatory Ligands are Upregulated in Amyotrophic Lateral Sclerosis

Receptor for Advanced Glycation End Products and its Inflammatory Ligands are Upregulated in Amyotrophic Lateral Sclerosis

Amyotrophic lateral sclerosis (ALS) is a fatal motor neuron disorder of largely unknown pathogenesis. Recent studies suggest that enhanced oxidative stress and neuroinflammation contribute to the progression of the disease. Mounting evidence implicates the receptor for advanced glycation end-product...

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Bibliographic Details
Main Authors: Judyta eJuranek, Gurdip eDaffu, Joanna eWojtkiewicz, David eLacomis, Julia eKofler, Ann Marie eSchmidt
Format: Article
Language:English
Published: Frontiers Media S.A. 2015-12-01
Series:Frontiers in Cellular Neuroscience
Subjects:
Amyotrophic Lateral Sclerosis
Rage
Spinal Cord
HMGB1
CML
S100B
Online Access:http://journal.frontiersin.org/Journal/10.3389/fncel.2015.00485/full

Internet

http://journal.frontiersin.org/Journal/10.3389/fncel.2015.00485/full

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