Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]

Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]

Bibliographic Details
Main Authors: Di Yang, Danjie Zhu, Shaoyu Zhu, Fabo Feng, Chen Gong, Chunmao Chen, Liang Chen
Format: Article
Language:English
Published: Elsevier 2019-04-01
Series:Biomedicine & Pharmacotherapy
Online Access:http://www.sciencedirect.com/science/article/pii/S0753332219308285
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spelling doaj-21dc38215dd8442282d3e239ff9f04e32021-05-20T07:37:51ZengElsevierBiomedicine & Pharmacotherapy0753-33222019-04-01112Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]Di Yang0Danjie Zhu1Shaoyu Zhu2Fabo Feng3Chen Gong4Chunmao Chen5Liang Chen6Department of Orthopaedic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, 215006, China; Department of Orthopaedic Surgery, Zhejiang Provincial People’s Hospital, Hangzhou, Zhejiang, 310014, China; People’s Hospital of Hangzhou Medical College, Hangzhou, Zhejiang, 310014, ChinaDepartment of Orthopaedic Surgery, Zhejiang Provincial People’s Hospital, Hangzhou, Zhejiang, 310014, China; People’s Hospital of Hangzhou Medical College, Hangzhou, Zhejiang, 310014, ChinaDepartment of Orthopaedic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, 215006, ChinaDepartment of Orthopaedic Surgery, Zhejiang Provincial People’s Hospital, Hangzhou, Zhejiang, 310014, China; People’s Hospital of Hangzhou Medical College, Hangzhou, Zhejiang, 310014, ChinaDepartment of Orthopaedic Surgery, The People’s Hospital of Bozhou, Bozhou, Anhui, 236800, ChinaDepartment of Orthopaedic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, 215006, ChinaDepartment of Orthopaedic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, 215006, China; Corresponding author.http://www.sciencedirect.com/science/article/pii/S0753332219308285
collection DOAJ
language English
format Article
sources DOAJ
author Di Yang
Danjie Zhu
Shaoyu Zhu
Fabo Feng
Chen Gong
Chunmao Chen
Liang Chen
spellingShingle Di Yang
Danjie Zhu
Shaoyu Zhu
Fabo Feng
Chen Gong
Chunmao Chen
Liang Chen
Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]
Biomedicine & Pharmacotherapy
author_facet Di Yang
Danjie Zhu
Shaoyu Zhu
Fabo Feng
Chen Gong
Chunmao Chen
Liang Chen
author_sort Di Yang
title Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]
title_short Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]
title_full Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]
title_fullStr Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]
title_full_unstemmed Corrigendum to “17β-Estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [Biomed. Pharmacother. 207 (2018) 1004–1009]
title_sort corrigendum to “17β-estradiol/extrogen receptor β alleviates apoptosis and enhances matrix biosynthesis of nucleus pulposus cells through regulating oxidative damage under a high glucose condition” [biomed. pharmacother. 207 (2018) 1004–1009]
publisher Elsevier
series Biomedicine & Pharmacotherapy
issn 0753-3322
publishDate 2019-04-01
url http://www.sciencedirect.com/science/article/pii/S0753332219308285
work_keys_str_mv AT diyang corrigendumto17bestradiolextrogenreceptorballeviatesapoptosisandenhancesmatrixbiosynthesisofnucleuspulposuscellsthroughregulatingoxidativedamageunderahighglucoseconditionbiomedpharmacother207201810041009
AT danjiezhu corrigendumto17bestradiolextrogenreceptorballeviatesapoptosisandenhancesmatrixbiosynthesisofnucleuspulposuscellsthroughregulatingoxidativedamageunderahighglucoseconditionbiomedpharmacother207201810041009
AT shaoyuzhu corrigendumto17bestradiolextrogenreceptorballeviatesapoptosisandenhancesmatrixbiosynthesisofnucleuspulposuscellsthroughregulatingoxidativedamageunderahighglucoseconditionbiomedpharmacother207201810041009
AT fabofeng corrigendumto17bestradiolextrogenreceptorballeviatesapoptosisandenhancesmatrixbiosynthesisofnucleuspulposuscellsthroughregulatingoxidativedamageunderahighglucoseconditionbiomedpharmacother207201810041009
AT chengong corrigendumto17bestradiolextrogenreceptorballeviatesapoptosisandenhancesmatrixbiosynthesisofnucleuspulposuscellsthroughregulatingoxidativedamageunderahighglucoseconditionbiomedpharmacother207201810041009
AT chunmaochen corrigendumto17bestradiolextrogenreceptorballeviatesapoptosisandenhancesmatrixbiosynthesisofnucleuspulposuscellsthroughregulatingoxidativedamageunderahighglucoseconditionbiomedpharmacother207201810041009
AT liangchen corrigendumto17bestradiolextrogenreceptorballeviatesapoptosisandenhancesmatrixbiosynthesisofnucleuspulposuscellsthroughregulatingoxidativedamageunderahighglucoseconditionbiomedpharmacother207201810041009
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