Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart Failure
Calcified atheromatous aortic lesion causing significant narrowing of the aorta is an uncommon clinical entity. This calcified atheroma leads to obstruction of the lumen of the aorta simulating acquired coarctation of aorta causing impaired perfusion of lower limbs, visceral ischemia, and hypertensi...
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doaj-1ace8af00ae34cf3bc12cd6bd95908212020-11-25T03:32:41ZengSAGE PublishingJournal of Investigative Medicine High Impact Case Reports2324-70962017-01-01510.1177/232470961668947710.1177_2324709616689477Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart FailureSajin Karakattu MD0Ghulam Murtaza MD1Sharma Dinesh MD2Kamesh Sivagnanam MD3Jeffrey Schoondyke MD4Timir Paul MD, PhD5East Tennessee State University, Johnson City, TN, USAEast Tennessee State University, Johnson City, TN, USAEast Tennessee State University, Johnson City, TN, USAEast Tennessee State University, Johnson City, TN, USAEast Tennessee State University, Johnson City, TN, USAEast Tennessee State University, Johnson City, TN, USACalcified atheromatous aortic lesion causing significant narrowing of the aorta is an uncommon clinical entity. This calcified atheroma leads to obstruction of the lumen of the aorta simulating acquired coarctation of aorta causing impaired perfusion of lower limbs, visceral ischemia, and hypertension. We report a case of 58-year-old patient who presented with dyspnea on exertion, orthopnea, paroxysmal nocturnal dyspnea, 25-lb weight gain, lower extremity edema, and chest pain. Extensive workup including computed tomography and magnetic resonance imaging revealed a large calcific mass in the aortic arch causing his presenting symptoms. After surgical correction his symptoms resolved. Any patient presenting with heart failure symptoms in the setting of uncontrolled renovascular hypertension, intermittent claudication symptoms, or visceral ischemia with normal ejection fraction but moderate to severe left ventricular hypertrophy should be in high suspicion for acquired coarctation of aorta. The routine thorough examination of pulses in bilateral upper and lower extremities in all hypertensive patients is a very simple and useful clinical tool to diagnose acquired aortic coarctation.https://doi.org/10.1177/2324709616689477 |
collection |
DOAJ |
language |
English |
format |
Article |
sources |
DOAJ |
author |
Sajin Karakattu MD Ghulam Murtaza MD Sharma Dinesh MD Kamesh Sivagnanam MD Jeffrey Schoondyke MD Timir Paul MD, PhD |
spellingShingle |
Sajin Karakattu MD Ghulam Murtaza MD Sharma Dinesh MD Kamesh Sivagnanam MD Jeffrey Schoondyke MD Timir Paul MD, PhD Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart Failure Journal of Investigative Medicine High Impact Case Reports |
author_facet |
Sajin Karakattu MD Ghulam Murtaza MD Sharma Dinesh MD Kamesh Sivagnanam MD Jeffrey Schoondyke MD Timir Paul MD, PhD |
author_sort |
Sajin Karakattu MD |
title |
Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart Failure |
title_short |
Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart Failure |
title_full |
Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart Failure |
title_fullStr |
Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart Failure |
title_full_unstemmed |
Supersized Atheroma Causing Acquired Coarctation of Aorta Leading to Heart Failure |
title_sort |
supersized atheroma causing acquired coarctation of aorta leading to heart failure |
publisher |
SAGE Publishing |
series |
Journal of Investigative Medicine High Impact Case Reports |
issn |
2324-7096 |
publishDate |
2017-01-01 |
description |
Calcified atheromatous aortic lesion causing significant narrowing of the aorta is an uncommon clinical entity. This calcified atheroma leads to obstruction of the lumen of the aorta simulating acquired coarctation of aorta causing impaired perfusion of lower limbs, visceral ischemia, and hypertension. We report a case of 58-year-old patient who presented with dyspnea on exertion, orthopnea, paroxysmal nocturnal dyspnea, 25-lb weight gain, lower extremity edema, and chest pain. Extensive workup including computed tomography and magnetic resonance imaging revealed a large calcific mass in the aortic arch causing his presenting symptoms. After surgical correction his symptoms resolved. Any patient presenting with heart failure symptoms in the setting of uncontrolled renovascular hypertension, intermittent claudication symptoms, or visceral ischemia with normal ejection fraction but moderate to severe left ventricular hypertrophy should be in high suspicion for acquired coarctation of aorta. The routine thorough examination of pulses in bilateral upper and lower extremities in all hypertensive patients is a very simple and useful clinical tool to diagnose acquired aortic coarctation. |
url |
https://doi.org/10.1177/2324709616689477 |
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