Cellular and Molecular Mechanisms in Pathogenesis of Multiple Sclerosis

Cellular and Molecular Mechanisms in Pathogenesis of Multiple Sclerosis

Multiple sclerosis (MS) is one of the most common neurological disorders in young adults. The etiology of MS is not known, but it is generally accepted that it is autoimmune in nature. Our knowledge of the pathogenesis of MS has increased tremendously in the past decade through clinical studies and...

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Bibliographic Details
Format: eBook
Language:English
Published: Basel, Switzerland MDPI - Multidisciplinary Digital Publishing Institute 2020
Subjects:
Medicine and Nursing
A971432
autoimmune disease
AUY954
bioinformatics
CD4+ T cells
central memory T cell
clonal analyses
demyelination
disease activity
effector memory T cell
experimental autoimmune encephalomyelitis
experimental autoimmune encephalomyelitis (EAE)
gliosis
glutamate synaptic dysfunction
granulocyte-macrophage colony-stimulating factor
histology
IDO
in utero electroporation
inside-out
kynurenic acid
kynurenine pathway
laquinimod
lesioned brain
lineage
lymphocytes
memory T cells
microglia
mitochondria
monocytes
morphometric analyses
multiple sclerosis
N-acetylserotonin
n/a
NAD+, multiple sclerosis
neuroinflammation
neutrophils
NG2-glia
NLR
oligodendrocytosis
outside-in
oxidative stress
ozanimod
pentamidine
pro-inflammatory cytokines
progenitors
quinolinic acid
relapsing-remitting experimental autoimmune encephalomyelitis
S100B
S1P1
S1P5
sphingosine-1-phosphate receptors
T lymphocytes
tissue-resident T cell
top-down proteomics
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